Community-Acquired Pneumonia Pathophysiology

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PATHOPHYSIOLOGY PREDISPOSING FACTOR PRECIPITATING FACTOR

- Gender (Male) - Polluted environment


- Age (35 y/o) - Cigarette smoking

entrance of bacteria in airway


material propelled into alveolar system (Streptococcus pneumonia, Haemophilus
influenza and Staphylococcus species)

adherence to alveolar macrophages exposure of WBC - 25.1 (H)


cell wall component (bacterial invasion to lungs)

loss of effectiveness of defense penetrate the sterile lower


acute inflammatory response affects alveoli
mechanism respiratory tract (lungs)

Acute pain related to inflammation


of lung parenchyma colonization multiplication

secondary to pleural effusion


multiplication of bacteria in release of damaging exotoxins
Irritation of airways
the urinary tract - cough
- crackles
infection - dyspnea
goblet cells mucus production
irritation of lining Hgb - 11.3 (H)
in urinary tract Hct - 33.9 (L)
Plt - 677 (H) inflammation
occludes airway Mgt:
occludes the airway
Removal of secretions.
Vasodilation and blood Adequate hydration of 2 to
Urinary Tract flow to area 3 liters per day.
airways constriction
Infection Humidification may loosen
impairing 02 and CO2
exchange secetions and improve
venilation.
S/S: Decrease O2 intake
- Left-sided alveolar leakage
chest pain
- chills
- SOB fluid leakage to pleural space

decreasing lung alveolar overinflates


expansion & burst
Chest x-ray Pleural effusion

air leaks to chest cavity

Ineffective airway clearance Ineffective breathing pattern related


related to pleuritic pain to decreased lung volume capacity lung collapse
secondary to pneumonia secondary to pleural effusion

Mgt: low blood oxygen levels in


Encourage avoidance of the body
overexertion.
Semi-Fowlers position to
promote rest and
breathing. multiple organs deprived
Maintenance of proper
fluid volume.
Maintenance of adequate
nutrition. DEATH
Administer analgesics as
prescribed.

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