This patient presented with gestational hypertension. Assessment findings included elevated blood pressure and general weakness. The nursing diagnosis was impaired tissue perfusion related to maternal hypovolemia. The plan was to monitor the patient and fetus closely through tests such as NSTs and biophysical profiles. Interventions focused on education, monitoring fetal activity and growth, assessing the fetus' response to stress, and administering corticosteroids if premature delivery was necessary to promote fetal lung maturity. The overall goal was to prevent progression of the hypertension and promote the best outcomes for both mother and baby.
This patient presented with gestational hypertension. Assessment findings included elevated blood pressure and general weakness. The nursing diagnosis was impaired tissue perfusion related to maternal hypovolemia. The plan was to monitor the patient and fetus closely through tests such as NSTs and biophysical profiles. Interventions focused on education, monitoring fetal activity and growth, assessing the fetus' response to stress, and administering corticosteroids if premature delivery was necessary to promote fetal lung maturity. The overall goal was to prevent progression of the hypertension and promote the best outcomes for both mother and baby.
This patient presented with gestational hypertension. Assessment findings included elevated blood pressure and general weakness. The nursing diagnosis was impaired tissue perfusion related to maternal hypovolemia. The plan was to monitor the patient and fetus closely through tests such as NSTs and biophysical profiles. Interventions focused on education, monitoring fetal activity and growth, assessing the fetus' response to stress, and administering corticosteroids if premature delivery was necessary to promote fetal lung maturity. The overall goal was to prevent progression of the hypertension and promote the best outcomes for both mother and baby.
This patient presented with gestational hypertension. Assessment findings included elevated blood pressure and general weakness. The nursing diagnosis was impaired tissue perfusion related to maternal hypovolemia. The plan was to monitor the patient and fetus closely through tests such as NSTs and biophysical profiles. Interventions focused on education, monitoring fetal activity and growth, assessing the fetus' response to stress, and administering corticosteroids if premature delivery was necessary to promote fetal lung maturity. The overall goal was to prevent progression of the hypertension and promote the best outcomes for both mother and baby.
Objective: Impaired Nursing care for PIH Do a nonstress Independent 1.Decrease in placental Patient blood flow results in demonstrates T: 37.5 Tissue involves providing test (NST), 1.Present information to reduced gas exchange normal CNS Perfusion adequate nutrition, Biophysical profile and impaired nutritional reactivity on P: 75 patient/couple concerning related to good prenatal care, and, Doppler flow functioning of the nonstress test R: 13 home assessment or placenta. Potential (NST) maternal and control of pre- studies to the noting daily fetal outcomes of poor BP: 140/80 hypovolemia existing hypertension check both the movements and when to placental perfusion in relation during pregnancy patient and the include a malnourished, Patient is free of -clammy skin seek immediate medical with decrease the baby LBW infant, and late deceleration - Pale skin attention. prematurity associated Intrauterine incidence and severity with early delivery, growth of preeclampsia. Early 2. Name factors affecting abruptio placentae, and Patient has no retardation recognition and fetal activity. fetal death. Reduced decrease in FHR Subjective: prompt treatment of fetal activity means fetal on contraction The patient felt 3. Report signs of vaginal compromise (occurs preeclampsia can stress nausea, and bleeding, uterine before detectable test/oxytocin generalized prevent progression tenderness, abdominal alteration in FHR and challenge test weakness to eclampsia. pain, and decreased fetal indicates demand for (CST/OCT). immediate activity evaluation/intervention.
4. Evaluate fetal growth; 2. Cigarette smoking,
measure progressive medication/drug use, serum glucose levels, fundal accompany growth environmental sounds, at each office visit or time of day, and sleep- periodically during stress wake cycle of the fetus home visits, as can increase or decrease fetal movement. appropriate. INTERVENTIONS RATIONALE 5. Check FHR manually or 3. Immediate attention and electronically, as indicated. intervention increases the likelihood of a positive outcome 6. Note fetal response to medications in direction of 4. Reduced placental the doctor such as MgSO4, functioning may accompany phenobarbital, and PIH, resulting in IUGR. Chronic diazepam. intrauterine stress and uteroplacental insufficiency Dependent decrease amount of fetal 7. Assess fetal response to contribution to amniotic fluid BPP criteria or CST, as pool. maternal status indicates. 5. Helps evaluate fetal well- 8. Assist with assessment being. An elevated FHR may of fetal maturity and well- show a compensatory response being using L/S ratio, to hypoxia, prematurity, or presence of PG, estriol abruptio placentae. levels, FBM, and sequential sonography beginning at 20–26 weeks’ gestation. INTERVENTIONS RATIONALE 9. Assist with assessment 6. In the event of declining maternal/fetal condition, of maternal plasma risks of delivering a preterm infant are weighed volume at 24–26 weeks’ against the risks of continuing the pregnancy, using gestation using Evans’ results from evaluative studies of lung and kidney blue dye when indicated. maturity, fetal growth, and placental functioning. IUGR is associated with reduced maternal volume and 10. Utilizing an vascular changes. ultrasonography, assist with assessment of 9. Identifies fetus at risk for IUGR or intrauterine fetal placental size. demise associated with reduced plasma volume and reduced placental perfusion. 11. Give corticosteroid (dexamethasone, 10. Reduced placental function and size are associated betamethasone) IM for at with PIH. least 24–48 hr, but not 11. Corticosteroids are thought to induce fetal more than 7 days before pulmonary maturity (surfactant production) and delivery, when severe PIH prevent respiratory distress syndrome, at least in a necessitates premature fetus delivered prematurely because of condition or delivery between 28 and inadequate placental functioning. Best results are 34 weeks’ gestation. obtained when fetus is less than 34 weeks’ gestation and delivery occurs within a week of corticosteroid administration