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    PERIPHERAL

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    tyulde
    The document discusses variations in arterial pulses and abnormal arterial and venous findings. It provides tables describing pulse characteristics, peripheral arterial disease, acute arterial occlusion, abdominal aortic aneurysm, Raynaud's phenomenon, chronic venous insufficiency, deep vein thrombosis, thrombophlebitis, lymphedema, and the differences between arterial and venous ulcers.

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    PERIPHERAL

    Uploaded by

    tyulde
    4 views2 pages
    The document discusses variations in arterial pulses and abnormal arterial and venous findings. It provides tables describing pulse characteristics, peripheral arterial disease, acute arterial occlusion, abdominal aortic aneurysm, Raynaud's phenomenon, chronic venous insufficiency, deep vein thrombosis, thrombophlebitis, lymphedema, and the differences between arterial and venous ulcers.

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    PERIPHERAL (1)

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    The document discusses variations in arterial pulses and abnormal arterial and venous findings. It provides tables describing pulse characteristics, peripheral arterial disease, acute arterial occlusion, abdominal aortic aneurysm, Raynaud's phenomenon, chronic venous insufficiency, deep vein thrombosis, thrombophlebitis, lymphedema, and the differences between arterial and venous ulcers.

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    © All Rights Reserved
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    4 views2 pages

    PERIPHERAL

    Uploaded by

    tyulde
    The document discusses variations in arterial pulses and abnormal arterial and venous findings. It provides tables describing pulse characteristics, peripheral arterial disease, acute arterial occlusion, abdominal aortic aneurysm, Raynaud's phenomenon, chronic venous insufficiency, deep vein thrombosis, thrombophlebitis, lymphedema, and the differences between arterial and venous ulcers.

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    © All Rights Reserved
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    of 2

    Table 13.1 Variations in Arterial Pulses (continued) Table 13.

    2 Abnormal Arterial Findings (continued)


    Pulse Characteristics and Causes
    Peripheral Arterial Disease Acute Arterial Occlusion
    Pulsus Paradoxus Characteristics: Palpable
    decrease in amplitude
    on quiet inspiration; with
    blood pressure cuff, systolic
    decreases of more than
    10 mm Hg during inspiration
    Plaque formation in peripheral It may result from
    Causes: Pericardial
    arteries limits oxygenated progression of PAD or
    tamponade, constrictive
    blood from reaching tissues. thrombus from another
    pericarditis, and obstructive
    Resulting ischemia causes source. In the latter case, a
    lung disease
    cramping pain (claudication) thrombus may break off and
    in the related area. Severe travel through the arterial
    occlusion may cause ulcers, system to a smaller vessel
    which turn may lead to that is then occluded.
    Table 13.2 Abnormal Arterial Findings gangrene and amputation.

    Abdominal Aortic Raynaud’s Phenomenon/


    Aneurysm Disease Table 13.3 Abnormal Venous Findings
    Chronic Venous Insufficiency Neuropathy

    Raynaud’s phenomenon
    is used when the cause is
    An aneurysm (outpouching
    attributed to a connective
    of an arterial wall) results
    tissue disorder. When the
    from a weakened or
    etiology is unknown (most
    damaged medial arterial Peripheral neuropathies are
    cases), it is called Raynaud’s Malfunctioning of the
    layer. It may occur in any most common in patients
    disease. Symptoms include unidirectional valves impairs
    artery but is most common with diabetes. Paresthesias,
    numbness, tingling, pain, blood return to the affected
    in the aorta. These critical burning sensations, and
    extreme pallor, cyanosis, and extremity. Causes are primary
    emergencies are often fatal. numbness may occur, along
    coolness of the hands. valvular incompetence (which with decreased senses of
    may be congenital), sequeale vibration, pain, temperature,
    (table continues on page 260) of DVT, or both. and proprioception. Daily
    foot assessment is critical.

    CHAPTER 13 Vascular, Lymphatics Assessment 259 260 UNIT 3 Regional Examinations


    Table 13.3 Abnormal Venous Findings (continued) Table 13.4 Arterial Versus Venous Ulcers
    Arterial Venous
    Thrombophlebitis Deep Vein Thrombosis
    Location Toes, metatarsals, Ankle, medial malleolus,
    malleoli, and heel and distal third of leg
    Borders Regular Irregular
    Ulcer base Pale, yellow Red, pink
    Drainage Minimal Moderate to large
    amount
    Gangrene May be present Not present
    Pain Painful; decreased Aching pain, feeling of
    with dependency heaviness; decreased
    DVT results from thrombus with elevation
    formation in deep veins. Skin Pale, inflamed, and Stasis dermatitis and
    Superficial thrombophlebitis They are more common necrotic pigmentation changes
    results from thrombus in the lower extremities.
    formation in the superficial Presenting symptoms are Pulses Decreased or Normal and may be
    veins. Assessment findings unilateral edema of the absent difficult to palpate
    are unilateral localized pain or extremity, redness, pain because of edema
    achiness, edema, warmth, and or achiness, and warmth.
    redness. In superficial veins, Unrecognized DVTs are
    a palpable mass or cord may responsible for most deaths
    also be present along the vein. from pulmonary emboli (PE).

    Lymphedema Lymphedema occurs when


    lymph channels or nodes
    are obstructed. Primary
    lymphedema is congenital.
    Secondary lymphedema
    results from injury, scarring,
    excision of lymph nodes,
    or, sometimes, trauma
    or chronic infection. As
    lymphedema progresses, the
    skin may thicken, redden,
    and show nonpitting edema.
    Small vesicles with lymphatic
    fluid may develop in more
    advanced stages. Cellulitis is
    a frequent complication.

    CHAPTER 13 Vascular, Lymphatics Assessment 261 262 UNIT 3 Regional Examinations

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