Medicowesome - Anatomy
Medicowesome - Anatomy
Medicowesome - Anatomy
The aim is to help the students who want to give upcoming NEET PG Exam and are clueless about how to plan and prepare ?
I know my friend struggling day in and out for life in Covid wards. Exams doesn't matter now.
When they will have time in hand I want to help them out with plan beforehand. They are already brilliant enough to have been through all the
challenges. :)
Enjoy learning! ^__^
I know you can't predict what is important for this or next exams. Atleast with time in hand you can glance through some topics that contribute
70% of any exam. I am sharing the list 1st year subject I made during my preparation.
1.ANATOMY
Embryology
1. Pharyngeal arches
2. Neural crest derivatives
3. Oogenesis and spermatogenesis
4. Notochord and remnants
5. Extraembryonic mesoderm
6. Diaphragm
7. Cardiovascular system ( Abnormal subclavian artery)
8. Urogenital system
Histology
1. Cerebellum
2. Osteoblasts
3. Urinary bladder epithelium
4. Cell junctions
5. Collagen types
6. cartilage
7. Tonsil,Lymph nodes, spleen,thymus
8. Stomach glands
9. Skin with sebaceous glands
10. Connective tissue types
11. Retina
12. Salivary gland
NEUROLOGY
1. Fornix
2. Corpus callosum
3. Cranial nerves and its lesion
4. Brainstem syndromes (lateral medullary )
5. Foramen of skull
6. Facial nerve and trigeminal nerve
7. blood brain barrier
8. Functional area and functional columns
9. Parasympathetic ganglion
10. Phrenic nerve and vagus
GROSS
Vitamin (Coenzyme)
Dentition And Xray of wrist elbow and pelvis to determine age Histology (25)
Concentration dependent kinetics and time dependent kinetics and post antibiotic effect Pediatrics (153)
Meningitis
Exanthematous disease
Managment of ptosis
► June (13)
► May (14)
Light reflex and accomodation reflex and pupil
► April (35)
3,4,6 Cranial nerve
► March (27)
OCT ,Fluorescein angiograhy,tonometers,charts,
► February (14)
Sudden loss of vision and gradual loss of vision differentials
► January (27)
3. ENT-
► 2019 (240)
Appearance of tympanic membrane in various disease
► 2018 (246)
Surgery (Tonsillectomy,Adenoidectomy,Mastoidectomy)
► 2017 (765)
Paranasal sinus
► 2016 (517)
CSF rhinorrhea
► 2015 (507)
Laryngeal disease (Papilloma,vocal cord paralysis, laryngeal muscles action, laryngeal cancers)
► 2014 (225)
Tracheostomy
► 2013 (102)
Abscess
► 2012 (31)
Nasopharyngeal carcinoma
► 2011 (14)
Juvenile angiofibroma
Otosclerosis
Pinterest
Cochlear implants
Medicowesome
Oral cavity cancers
Pre-eclampsia (Definitions)
Abortions
Follow by Email
Recurrent pregnancy loss (Investigations and causes)
MTP
Rh incompatibility
Amenorrhea (primary and secondary) Followers
Mullerian anomalies (Class)
Followers (133) Next
Asherman, AIS,Gonadectomy indication
PCOS -Diagnosis,drugs
Endometriosis
Cervical,vulval,ovarian,endometrial cancer,Fibroid
Krukenberg
PID Follow
Prolapse
Emergency contaceptives
IUD
Female sterilisation
2.PEDIATRICS-
Developmental milestones
Neonatal reflexes
Neonatal resuscitation
Congenital infections-TORCH
Diarrhea,pneumonia,dehydration managment
Pediatric epilepsy
Rickets
meningitis
Nephrotic syndrome
Trisomies
3. ANESTHESIA-
Vaporisers color
Inhalational agents
Monitoring (Capnography)
Circuits
Airway devices
Modes of ventilation
4.DERMATOLOGY-
Pigmentation Diseases
1. Hyperpigmentation
2. Nevus (Nevus of ota/mongolian spot/CMN/AMN)
3. Melasma
4. Acanthosis nigricans
5. Becker nevus
6. Hypopigmentation -PKDL/Pityriasis versicolor/alba/Hansens
7. Depigmentation-Vitiligo/Contact leukoderma
Treatment of Psoriasis
Blistering disease
1. Pemphigus(clinical treatment)
2. Bullos pemphigoid
3. dermatitis herpetiformis
4. Histopathology of blister level and Direct immunofluorescence image
Infections
Acne,Rosacea
5.RADIOLOGY-
CNS tumor
Bone tumor
BIRADS
TIRADS
Radiotherapy basics
Doppler waveforms
6.ORTHOPEDICS-
Shoulder dislocation
Infections(TB,Osteomyelitis)
CTEV
AVN
Pseudoarthrosis
malunion
Instruments
7.SURGERY-
Burn
Triage
Trauma
Incisions,suture,foleys,NG tube,Knots,Scores
Bariatric surgery
Aortic aneurysm
Meckel diverticulum
Wound classification
Cancer-(Breast,rectum,stomach,oesophagus,HCC,prostate,thyroid)
8.MEDICINE-
ECG visuals
Stroke
Meningitis (Bacterial,viral,aseptic)
Seizure
Electrolyte imbalance
ABG
Pericardial d/o
hepatitis(Viral,autoimmune)
Glomerular disease
UTI
CKD
GBS
Poisoning
SIADH, DI,Pheochromocytoma
IBS,UC ,Crohns
Cardiomyopathy
dyslipidemia management
10.PSYCHIATRY-
MMSE
Psychotic disorders
1. Schizophrenia
2. Delusional d/o- Named syndromes (Capgras,Fregoli,Othello,Ekbom)
1. Mania
2. Depression
3. Bipolar
Neurotic disorder
1. Anxiety disorder
2. OCD related disorder
3. Dissociative disorder
4. Trauma and stress related disorder
5. Somatoform disorder
Sleep disorder
Eating disorder
Sexual disorder
Defence mechanism
Read more »
PS : Question which was asked in central institute examination (I felt it should be mentioed here)
Which ligament is felt while giving pudendal block?
Sacrospinal ligament.
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That's it
-Demotional bloke
That's it
- Demotional bloke
Hello Awesomites !
-Upasana Y.
Integrating Trendelenburg
This blog will compel all the signs /symptoms Or test related to 'Trendelenburg'. Let us know if you know more of the 'Trendelenburg' in the
comment section so we can integrate it here.
Trendelenburg's gait:
A child with unilateral dislocation of hip lurches on the affected side while bearing weight on it. Seen in DDH, poliomyelitis, Superior Gluteal nerve
palsy.
Trendelenburg's gait is also known as Abductor gait or lurching gait.
Trendelenburg's test:
Trendelenburg's surgery/procedure:
It is done in GSV and SFJ incompetence. Here flush ligation of SFJ is done. Flush ligation means vein is ligated as close as possible.
Six tributaries also need to be ligated to reduce recurrence rate.
Laterally: Superior circumflex iliac.
Superior epigastric vein.
Medially: Superior external pudendal
Deep external pudendal
Distally: Accessory anterior saphenous vein
Posterior medical thigh vein
Stripping is an additional surgery. Should be done till knee, not below knee to avoid Saphaneous nerve.
It is done to determine the incompetency of the sapheno-femoral valve and incompetency of the communicating vein.
In both the tests, patient is first placed in the recumbent position and his legs are raised to empty the veins. The sapheno-femoral junction is now
compressed with the thumb or a torniquet can be used.
1) To check sapheno-femoral valve incompetency, patient is asked to stand up quickly and pressure is released. If varices fill quickly by a column
of a blood from above, it indicates incompetency of the sapheno-femoral valve. This is called Trendelenburg test.
2) To test communicating system, pressure is not released but maintain for 1 min. Gradual filling of the veins indicates incompetency of
communicating veins.
That's it
-Demotional bloke.
Horner Syndrome
Horner syndrome is a classic neurologic syndrome whose signs include miosis, ptosis, and anhidrosis.
NEUROANATOMY - Horner syndrome can result from a lesion anywhere along a three-neuron sympathetic pathway that originates in the
hypothalamus:
●The first-order neuron descends caudally from the hypothalamus to the first synapse, which is located in the cervical spinal cord (levels C8-T2,
also called ciliospinal center of Budge).
●The second-order neuron travels from the sympathetic trunk over the lung apex. It then ascends to the superior cervical ganglion, located near
the bifurcation of the common carotid artery.
●The third-order neuron from superior cervical ganglia then ascends within the adventitia of the internal carotid artery, through the cavernous
sinus. In the orbit and the eye, the oculosympathetic fibers innervate the iris dilator muscle as well as Müller's muscle, a small smooth muscle in
the eyelids responsible for a minor portion of the upper lid elevation and lower lid retraction.
First-order syndrome - Lesions of the sympathetic tracts in the brainstem or cervicothoracic spinal cord can produce a first-order Horner
syndrome.
The most common causes are:
(a)occlusion of PICA, which produces Horner syndrome as part of the Wallenberg syndrome.
(b)Brown-Séquard syndrome above T1, patient may present with ipsilateral Horner syndrome due to damage of oculosympathetic pathway.
Second-order syndrome — Second-order or preganglionic Horner syndromes can occur with trauma or surgery involving the spinal cord, thoracic
outlet, or lung apex.Other causes include pancoast tumor involving the lung apex.
Third-order syndrome — Third-order Horner syndromes often indicate lesions of the internal carotid artery such as an arterial dissection,
thrombosis, or cavernous sinus aneurysm
CLINICAL FEATURES -The classic signs of a Horner syndrome are ptosis, miosis, and anhidrosis.
1)The ptosis occurs as a result of paralysis of the Müller's muscle.
2)The degree of anisocoria is more marked in the dark than in light.
3)Anhidrosis is present in central or preganglionic (first- or second-order) lesions because the sympathetic fibers responsible for facial sweating
branch off at the superior cervical ganglion along the external carotid artery and its branches.
4)Horner syndrome is also a common feature of cluster headache.
SOURCE-UpToDate, Kaplan.
-Srikar Sama.
Posted by Dr. Srikar Sama 1 comment:
Feedback: Interesting (0) Kissable (0) Awesome (0) Nice (0)
Note: Don't use this diagram when you are studying squint. This diagram is used only for movements of eye muscles.
So every muscle has 3 actions except medial and lateral rectus.
Actions are divided into three types Primary, secondary and tertiary.
We know the basic or primary action of superior and inferior rectus is to elevate and depress the eyeball respectively.
According to the diagram given you also know that superior rectus can intort and adduct. Similarly, inferior rectus can extort and adduct the
muscles.
Now comes to SO(Superior oblique) and IO(Inferior oblique). Now this is also basic which we fail to notice often. These muscles are coming to
tarsus part of eye obliquely. So, primary action is intortion and extortion respectively. Now go to diagram and you will notice that IO actually
corresponds to SR hence it performs elevation and SO corresponds to IR so it performs depression.
Also, both the muscles are "Abductors"
-Demotional bloke
Today we will study Inguinal Lymph nodes along with its clinical significance.
The Lymph nodes lying above it is called as superficial inguinal Lymph nodes.
The Lymph nodes lying beneath it is called as deep inguinal Lymph nodes.
The superficial Lymph nodes are further divided into horizontal and vertical groups.
The deep lymph nodes are deep to fascia lata and 1-2 nodes can be found in femoral canal. This nodes
in the femoral canal are called as deep inguinal Lymph node of Rosenmuller or of Cloquet. This
drains glans penis and clitoris.
Through the umbilicus is taken a watershed line of Lymphatic drainage of anterior abdominal wall.
The anterior abdominal wall below the umbilicus drains in the superficial inguinal lymph nodes.
Entire perineum except glans penis and glans clitoris are drained by superficial lymph nodes.
Most of the lower limb is drained by superficial inguinal lymph nodes except the territory of short
saphenous vein (This part of lower limb is drained by popliteal group of lymph nodes).
Some part of Uterus and uterine tubes are also drained by superficial inguinal lymph nodes.
Anal canal below pectinate line also drains in superficial inguinal lymph nodes.
CLINICAL SIGNIFICANCE:
In sexually transmitted diseases, you will find enlargement of this lymph nodes.
Inguinal lymph nodes are the frequent sites of metastasis for malignant lymphoma, squamous cell
carcinoma of anal canal, vulva and penis, malignant melanoma and squamous cell carcinoma of skin
over lower extremities or trunk.
-Upasana Y. :)
Neural Crest Cells are very special cells that form at around 2-3 weeks of gestation.
They're right next to the Neural folds of the Neural tube to begin with and then eventually come to lie lateral to the tube, after which they migrate
to various parts of the body giving rise to a bunch of things.
It's kind of SUPER IMPORTANT to memories what structures arise out of the Crest cells.
One of the reasons for it is that neural crest cell tumors will express certain neuroendocrine markers and that will make it easier to detect them on
histopathology.
The other reason is that it's literally the most favorite embryology question - whether it's NEET PG or Step1!
So here goes :
B - Branchial Arches
A - Aortopulmonary windows + Endocardial Cushions
B - Bronchogenic cells - Pneumocytes
Y - Yo
This will also help you understand why Melanomas , Bronchogenic Carcinoma , Schwannoma, Pheochromocytoma and even Neuroblastoma are
all positive for similar markers and have similar appearances at times along with similar histopathological fetaures.
~ A.P. Burkholderia
The femoral triangle is a subfascial space bounded superiorly by the inguinal ligament, medially by the adductor longus muscle, and laterally by
the sartorius muscle.
Introduction to neuroanatomy
This video was made by our Medicowesome Student Guest Author, Salman!!!!
Introduction to neuroanatomy
Read more »
NERVE SUPPLY :
(beta 2 and alpha 1 are adrenergic receptors of SANS ; muscarinic type 3 is a cholinergic receptor of PANS)
SANS inhibits micturition while PANS facilitates micturition. You don’t want to pee when you’re running a 100m sprint, an SANS-
dominant activity but you can comfortably pee at rest, a PANS-dominant activity.
Sensory fibres of pudendal nerve tell your CNS when the bladder is full. The motor fibres of pudendal nerve maintain EUS tonic
contraction by default so that you’re not always peeing.
The reflex arc, after higher centre commands, causes voluntary micturition by inhibiting the “contraction-effect” of motor fibres of
pudendal nerve.
To oversimplify matter (so that it’s easy to understand and remember): Level 2 control inhibits reflex arc. Level 3 control facilitates
reflex arc, causing micturition at will, once the bladder is full.
CLINICAL CORRELATES:
CORTICAL BLADDER
Associated with:
Multiple Sclerosis
Parkinson’s disease
Stroke, among others.
2 subtypes are:
Motor Paralytic bladder – Motor (efferent) pathway is damaged. However, patient can sense bladder fullness, resulting in prompt
diagnosis. Associated with:
Sensory Paralytic bladder – Sensory (afferent) pathway is damaged and hence, patient canNOT sense bladder fullness, resulting in
delayed diagnosis. Associated with:
-- Diabetes mellitus
-- Syringomyelia
-- Tabes dorsalis
Medicine pearl: When we talk about bladder pathology, we only refer to PANS (lesions above/ at/ below it) as SANS lesions
doNOT cause bladder pathology per se. However, bilateral lesion to L1 causes retrograde ejaculation and hence, infertility.
Hope this helps! Let me know if anything needs clarification. Happy studying!
-- Ashish Singh.
Maxillary artery is divided into three branches. Again, each branch is subdivided.
We also have to learn their course which is very confusing and we forget it during our exam :(
So, I came up with an easy way to learn it. I decided that I will show the course of the artery in form of a diagram ( you will get more marks! ) and
write down what it supplies.
Mnemonic for the Ascending tracts & Descending tracts in Spinal Cord
Hiiiii everyone, it’s mnemonic time again! From the title, I guess you all know what it is about… I had a hard time memorizing these tracts so I
created this mnemonic to help you guys out!
That Los Angeles STudent went up to stage and SCream using the PA system because he got a
Freaking Cool First Grade result.
Please comment if you have a better mnemonic! Let’s share our mnemonics ^.^
-Calvin Ong K. Y.
Helloooo everyone!!
Here’s some mnemonic I created myself for the branches of external carotid artery and the branches of facial artery. To memorize the name of
the branches of external carotid artery, you just need to....
Read more »
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