Tests in Pediatric Neurology
Tests in Pediatric Neurology
Tests in Pediatric Neurology
8//// The following drugs are recommended for the treatment of absences:
// Succinimides and Valproic acid
/// Hidantoins
/// Barbiturates
/// Carbamazepine
9//// Lennox-Gastaut Syndrome is defined as:
// Frequent, sometimes serial polymorphic seizures with mental retardation.
/// Only febrile seizures
/// Only partial seizures
/// Disease with benign course
//// The following drugs are recommended for the treatment of West syndrome:
// Hormones and Valproic acid or Vigabatrine
/// Phenobarbital
/// Diphenine
/// Diazepame
//// Generalized epilepsies are the following, except:
/// Absences
/// Myoclonic seizures
/// Tonic-clonic seizures
// Jacksons motor seizures
//// Partial seizures are the following, except:
/// Idiopathic ( age-related)
/// Symptomatic
/// Cryptogenic
// West syndrome
//// EEG pattern of typical absences is:
/// Temporal sharp waves
// 3 Hz. Spike-wave complexes
/// High amplitude generalized activity
/// 5-6 Hz. Spike-wave complexes
//// The typical EEG pattern of West syndrome is:
// Hypsarrhythmia
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//// The recommended daily dosage of ACTH for infantile spasm is:
/// 20-30 unit
// 80-100 unit
/// Less than 10 unit
/// 5 unit
//// The initial dosage of Prednisolone used for the treatment of infantile spasms is:
// 2 mg/kg/d
/// 0,5 mg/kg/d
/// 4 mg/kg/d
/// 1 mg/kg/d
//// The reduction of dosage of hormones in infantile spasms can be started:
/// When seizures become rare
// After cessation of seizures and hypsarrhythmia on EEG
/// After improving the psychomotor functioning
/// After improving the EEG patterns
//// The drug used in infantile spasms is:
// High doses of Piridoxine
/// Vitamin C
/// Vitamin D
/// Vitamin E
.//// The drug not recommended for the treatment of infantile spasms is:
/// ACTH
/// Immunoglobulin
/// Piridoxine
// Nivalin
//// The drug not recommended for the treatment of infantile spasms is:
/// Nivalin
// ACTH
/// Phenobarbital
///Piracetam
41 The complex absence is not characterized by:
// Abrupt onset
/// Tonic component
/// Clonic component
/// Gradual onset
//// The drug not recommended for the management of epileptic status is:
/// Diazepame or Lorazepam
/// Phenitoine
// Vigabatrine
/// Phenobarbitale
//// The drug administration in epilepsy lasts for:
/// 1-3 months
/// 3-6 months
// 3-5 years
/// 1 years
58//// The anticonvulsive therapy is conducted:
/// As a short period course for 1-2 years
// Permanently, with administration of drug for several years
/// By using of broad spectrum anticonvulsant for 6 months
/// By administration of drug for 1 year
59//// The anticonvulsive therapy is recommended to start by:
// Single anticonvulsant
/// Two or three anticonvulsants
/// Vitamins
/// Antibiotics
60 //// The drugs of first choice in febrile seizure are the following:
/// Phenobarbitale
// Valproic acid
/// Lamotrigine
/// Topiramate
61//// The drug recommended to use for the treatment of partial seizures is:
// Carbamazepine
/// Succinimide
/// B group vitamins
/// Hormone
62//// The drug not recommended to use for the treatment of partial seizures is:
/// Carbamazepine
/// Valproic acid
// Succinimnides
/// Phenobarbitale
63 The drug recommended to use for the treatment of absence seizures is:
/// Carbamazepine
/// Phenobarbitale
// Valproic acid
/// Phenitoine
64 The drug not recommended to use for the treatment of is:
/// Succinimides
/// Clonazepame
/// Valproic acid
// Carbamazepine
65 The following drugs are used for the treatment of Lennox-Gastou syndrome:
// Lamotrigine
/// Phenobarbitale
/// Piridoxyne
/// Antibiotics
66 The drugs not used for the treatment of Lennox-Gastaut syndrome is:
/// Monotherapy with Lamictal
// Phenobarbitale
/// Valproic acid
/// Clonazepame
67 The anticonvulsants of new generation is:
/// Phelbatole
/// Oxcarbamazepine
/// Acetazolamide
// Phenobarbitale
//// The basic anticonvulsant is not:
// Gabapentine
/// Phenobarbitale
/// Carbamazepine
/// Valproic acid
69 The basic anticonvulsant is :
/// Topiramate
/// Vagabatrine
// Carbamazepine
/// Midazolam
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70 Besides the drugs the approach also used for the treatment of intractable epilepsy is:
/// Physiotherapy
// Ketogenic diet
/// Occupational therapy
/// Psychotherapy
//// For the treatment of intractable epilepsy is used:
/// Electrophoresis
/// Occupational therapy
// Stimulation of Vagus
/// Psychotherapy
72 For the treatment of intractable epilepsy is used:
// Neurosurgical operations on brain
/// Occupational therapy
/// Psychotherapy
/// Electrophoresis
73 Porencephaly often causes the following form of cerebral palsy:
/// Spastic diplegia
/// Spastic tetraparesis
// Hemiparetic form
/// Hyperkinetic form
74 Aplasia of basal ganglia and nuclear jaundice cause the following form of cerebral palsy:
// Hyperkinetic form
/// Hemiparetic form
/// Spastic tetraparesis
/// Spastic diplegia
75 Littly disease is presented as the following form of cerebral palsy:
/// Spastic tetraparesis
// Spastic diplegia
/// Hyperkinetic form
/// Atonic-astatic form
76 The disorders of extremities in spastic diplegia are expressed:
/// On one side
// Disorder is symmetric
/// Upper extremities are affected more
/// Upper and lower extremities are affected on one side
//// In cerebral palsy the symmetric reflex from head to trunk and extremities is:
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/// Increased
// Is increased in neonate period and inhibited in pre-school period
/// Is absent
/// Is always changed
78 In cerebral palsy of paralitic form the muscle atrophy is expressed by the following ways:
/// Is absent
/// Occurred only in later residual period
/// Occurred only during muscle hypotonia
// Is mildly expressed and has diffuse manifestation
79 The prevalence of cerebral palsy per 1000 is:
/// 0,5 and less
// 1,5 and 2
/// 5 and more
/// 10 and more
80 In cerebral palsy of spastic tetraplegia form is expressed:
// Tetraparesis more affected the upper extremities
/// Hemiplegia on one side and hemiparesis on other side
/// Tetraparesis occurred only in severe cases
/// Disorders only in lower extremities
81 In cerebral palsy of tetraplegia form the muscle tone is:
/// Always decreased
/// Slightly increased
// Is increased significantly
/// Is increased only in later residual period
2//// In cerebral palsy of spastic diplegia form are affected:
/// Only internal capsule on one side
/// Cerebral peduncles
// Cerebral hemispheres, especially cortex
/// Brainstem
83 In cerebral palsy of spastic tetraplegic form patient start walking:
// In very rare cases
/// On the third year of life
/// After 5-6 year of age
/// Only in the case of intact mental field
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a.
b.
107 The inherited unconditioned reflexes of neonates disappears at the following ages:
/// Before 1 month
/// Before 1-2 months
// Before 3-5 moths
/// After 1 years
//// Landau inherited reflex in neonates disappears:
/// At 1 month of age
/// At 1-3 months of age
/// At 6 months of age
// It never disappears
109 The following are not TORCH infection:
// Rubella infection
/// Cytomegalovirus infection
/// Herpes Simplex infection
/// Toxoplasmosis
//// In congenital rubella infection microcephaly occurs:
/// In 100% of cases
// In 10-20% of cases
/// In 50% and more
/// It never occurs
111 Hydrocephaly, enophthalmy, spastic paresis, epileptic seizures, chorioretinitis associated
with fetal hepatitis in neonates are the features of:
/// Rubella infection
// Toxoplasmosis
/// Cytomegalovirus infection
/// Listeriosis
//// The most frequent form of developmental defect of brain is:
/// Myelocele
/// Macrocephaly
// Microcephaly
/// Dysostosis of skull and face
113 Microcephaly is divided by the following ways:
// Primary and secondary
/// Congenital and acquired
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/// C5-C6
/// C7-C8
/// D1-D2
// C5-D1
//// The obstetrical paralysis of hand of proximal type occurs during the damage of the following
segments:
// C5-C6
/// C7-C8
/// D1-D2
/// C5-D1
130 The obstetrical paralysis of hand of distal type ( Dejerin-Klumpke) occurs during the
damage of the following segments:
/// C5-C6
/// C7-C8
// D1-D2
/// C5-D1
131 The structural defect of the brain in cerebral palsy affects the following functions:
/// Only motor development
/// Only speech development
// The development of the whole brain
/// It doesn't affect on brain development
//// The cerebral palsy in relation to hypoxic-ischemic encephalopathy is:
/// Independent disease
/// Ordinary outcome
/// The have no relation
// One possible variant of outcome
133 In cerebral palsy of spastic diplegia form occurs:
/// Upper spastic paraparesis
/// Spastic hemiparesis
// Lower paraparesis
/// Tetraparesis
//// The spastic tetraparesis form of cerebral palsy is defined as:
/// Tetraparesis with mild mental retardation
// Tetraparesis with severe mental retardation
/// The peripheral paresis in hands and spastic paresis in legs
/// The spastic paresis in hands and peripheral paresis in legs
135 In cerebral palsy of atonic-astatic form are impaired the following structures:
/// Both internal capsules
/// Basal ganglias
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// Cerebellum
/// The brain hemispheres
//// The possible reason of traumatic injury of facial nerve in neonates is not:
/// Face presentation
/// Difficult labor
/// The fractures of temporal bone
// Subarachnoidal hemmorage
//// The reason of spinal cord trauma can be:
// Pelvic presentation
/// Cross birth
/// Normal cephalic presentation
/// Brow presentation
//// The spinal injury in neonates occurs in the following cases:
/// Intrauterine hypoxia
/// Natal period asphyxia
/// Natal period infection
// Natal period trauma
//// The severe damage of CNS can be defined by Apgar's scale by the following scores:
/// 0-1
// 1-4
/// 5-6
/// 6-7
140 The moderate damage of CNS can be defined by Apgar's scale by the following scores:
/// 0-1
/// 1-4
// 5-6
/// 6-7
//// The mild damage of CNS can be defined by Apgar's scale by the following scores:
/// 0-1
/// 1-4
/// 5-6
// 6-7
142 The etiological factor of hypoxic-ischemic encephalopathy is not:
/// Hypoxia
/// Trauma
/// Toxic process
// Congenital factor
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/// Traumatic
/// Toxic
/// Hypoxic
// Congenital
151 Dilatation of subarachnoid spaces occurs in the following forms of hydrocephaly:
/// Internal
// External
/// Basal
/// Convexital
//// Dilatation of brain ventricles and subarachnoid spaces occurs in the following forms of
hydrocephaly:
/// Internal
/// External
// Open
/// Convexital
153 Dilatation of only ventricles occurs in the following forms of hydrocephaly:
// Internal
/// External
/// Basal
/// Convexital
//// By the period of development hydrocephaly can be divided in the following forms:
/// Primary and secondary
// Congenital and acquired
/// Pre- and postnatal
/// Early ( before 6 month) and late ( after 6 month)
//// Passive hydrocephaly occurs in the following cases:
// Brain atrophy or progressive sclerosis of brain
/// Disorder of cerebrospinal fluid absorption
/// Venous stasis
/// Excessive production of cerebrospinal fluid
156 The basic sign of hydrocephaly is:
/// Increasing of intracranial pressure
// Imbalance between the production and absorption of cerebrospinal fluid
/// Dilatation of ventrical system and cerebrospinal circulation ways
/// Increased head circumference
//// The clinical feature of hypertensive-occlusive crisis implies:
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Nana Geladze
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